PM10 particles thicken the blood and boost inflammation, according to the results of an experimental study.
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Breathing diesel exhaust at levels typically found in large cities for as little as an hour can disrupt important blood vessel functions, suggesting a possible mechanism for increased heart attack rates during periods of high air pollution.
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Researchers exposed 23 healthy nonsmoking adults to concentrated ambient fine particles and fine particles plus ozone during 2-hour exposures.
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A panel study was conducted to measure the early physiological reactions characterized by blood biomarkers of inflammation, endothelial (the cells that line blood vessels) dysfunction, and coagulation in response to daily changes in air pollution in Erfurt, Germany.
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African-American and Latino children on corticosteroid asthma maintenance medication or with upper respiratory infections are adversely affected by current levels of air pollution, according to a study of primary school age children with asthma in Detroit.
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A time-series study of a very large database of 4 million emergency department visits to 31 hospitals in Atlanta has shown positive relationships between various air pollutants and respiratory disease.
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A large-scale epidemiological study of respiratory hospital admissions in children was carried out in the largest cities in Australia and New Zealand: Brisbane, Canberra, Melbourne, Perth, Sydney, Auckland and Christchurch.
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This multi-city study examined the association between PM10 and emergency hospitalization for heart attacks among elderly residents of 21 U.S. cities.
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PM10 concentrations below current EPA standards are associated with an increased rate of hospital admissions for congestive heart failure, in a study of seven U.S. cities.
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Particulate air pollution (PM10) increases the risk for ischemic strokes — those caused by a blood clot — according to a study by Harvard University researchers.
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